| What! … no potatoes? - Creation Magazine |
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What! … no potatoes?Governments are waking up to the need to preserve the ‘wild’ varieties of our food plants, with their rich stores of information. A highly qualified plant scientist tells us how this highlights the fallacy of evolution. Why are there so many people of Irish descent in North America and Australia? It harks back to the Irish Potato Famine of the 1840s. Over 1.5 million people died in Ireland when the potato crops failed due to a disease known as potato blight. Many people emigrated.
Therefore the crops in Europe were all susceptible to the disease when it arrived. (Ireland suffered the most because of its very high dependence on potatoes for the complex carbohydrate portion of their diet, whereas others had more grain crops). They succumbed because of the lack of genetic variety, which included the genes for resistance to blight. The pattern has been repeated many times since. In 1970 in the U.S., genetic uniformity resulted in loss of almost a billion dollars worth of maize because 80% of the varieties being grown were susceptible to a virulent disease known as ‘southern leaf blight.’1 Too successful?Plant breeders have been very successful in increasing the yields of all sorts of crop plants—so successful that farmers have been replacing the local, traditional varieties with the new varieties. For example, in China, at least 9,000 varieties of wheat have been lost since 1949. The ‘Green Revolution’ saw the development of high-yielding rice and wheat varieties and their rapid replacement of traditional, community-bred varieties (‘landraces’). For example, by 1984 in Bangladesh, 96% of the wheat grown consisted of Green Revolution varieties. A single variety of the ‘miracle wheat’ accounted for 67% of all the wheat planted.2 This has contributed to the feeding of many millions of people. However, the loss of the traditional varieties, and the reliance on relatively few new varieties, poses problems. Problems
Large areas of a uniform variety are susceptible to new strains of pests and disease for which the variety lacks resistance. These new pest or disease strains can be introduced from overseas, or new varieties can occur through normal reproduction which results in new combinations of existing genes. Just as with antibiotic resistance, these new disease strains do not arise through the development of new, functional genes,3 so this has nothing to do with particles-to-people evolution. To try to keep ahead of new strains of pests and diseases, plant breeders introduce new genes from wild plants of the crop species, or from ‘landraces,’ into new varieties. New varieties generally last only five to seven years before they are replaced. However, with loss of the wild types and landraces, plant breeders could lack the sources of genes for the further breeding needed to increase yields, decrease dependence on fertilizers and pesticides, improve quality, breed for drought resistance, cold/heat tolerance, salt tolerance, and many other things. So the loss of the genetic information needed to achieve these objectives is a serious problem. The U.N.’s Food and Agriculture Organisation (FAO) estimates that about 75% of genetic diversity in agricultural crops has been lost this century—largely by the replacement of landraces with the new varieties. Authorities are beginning to respond to this problem—see banking on genes. Denying evolutionMany scientists believe the dogma that the blind, purposeless forces of evolution (random mutations and natural selection) created all the genetic information in plants. Yet the (belated) push to preserve the wild varieties of our food plants highlights the fact that no amount of selection (artificial, by breeders, or natural) can generate information which is not there!
If random copying mistakes (mutations) originally generated all the information, surely it should not be too hard for highly intelligent scientists to create the required genes for breeding new improved varieties? However, with all that we now know about genes, no one can yet create a gene—for example, for rust resistance—from scratch.7 Plant breeders recognize that the information in the genes of plants is irreplaceable. The evolutionist E.O. Wilson wrote: ‘Each species is the repository of an immense amount of genetic information. The number of genes range from about 1,000 in bacteria and 10,000 in some fungi to 700,000 or more in many flowering plants and a few animals … . If stretched out fully, the DNA [in one cell] would be roughly a meter long. But this molecule is invisible to the naked eye. … The full information contained therein, if translated into ordinary-size letter of printed text, would just about fill all 15 editions of the Encyclopædia Britannica published since 1768.’8 Biologist David Janzen, University of Pennsylvania, said that destroying tropical forests for paper manufacture would be ‘like pulping the Library of Congress to get newsprint.’8 Just as the information in books comes from an intelligent source, so the information in the genes of living things also comes from an intelligent source. This source is clearly far more knowledgeable and intelligent than we who cannot yet create the genetic information ourselves and so we have to be concerned about the loss of genetic diversity. In their concern for the loss of this diversity, plant breeders agree that the genetic information is irreplaceable, and tacitly admit that it did not arise through random, non-intelligent processes, and that selection cannot re-create it, once lost. Faith that the blind forces of ‘evolution’ created all the genetic information is indeed a blind faith.
References and notes
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Why did the potatoes succumb to the disease? Potatoes came from the Andes mountains of South America, where many different varieties were grown, including some which could resist potato blight disease. When potatoes were introduced to Europe in the 1500s, this did not include varieties with resistance to this disease.
Why don’t strawberries taste like Dad’s used to?